PromaCare-TA / Tranexamic Acid

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PromaCare-TA yog cov tshuaj siv dav dav, tus neeg sawv cev antifibrinolytic tseem ceeb hauv WHO daim ntawv teev npe. Nws tau raug siv los ua cov tshuaj hemostatic.Nws yog ib qho tshuaj rau inhibition ntawm plasminogen rau plasmin hauv cov ntshav. Tranexamic acid sib tw inhibits kev ua kom plasminogen (ntawm kev khi rau lub kringle domain), yog li txo qis kev hloov pauv ntawm plasminogen rau plasmin (fibrinolysin), ib qho enzyme uas degrades fibrin clots, fibrinogen, thiab lwm yam plasma proteins, nrog rau cov procoagulant yam V thiab VIII. Tranexamic acid kuj ncaj qha inhibits plasmin kev ua haujlwm, tab sis ntau dua yuav tsum tau txhaj tshuaj ntau dua li qhov xav tau los txo qis kev tsim cov plasmin.


Product Detail

Khoom cim npe

 

Lub npe lag luam PromaCare-TA
CAS 1197-18-8 ib
Khoom npe Tranexamic Acid
Chemical Structure
Daim ntawv thov Tshuaj kho mob
Pob 25kgs net ib nruas
Qhov tshwm sim Dawb los yog yuav luag dawb, crystalline zog
Assay 99.0-101.0%
Solubility Dej soluble
Txee lub neej 4 xyoo
Cia Khaws lub thawv kaw kom nruj thiab nyob rau hauv qhov chaw txias. Khaws kom deb ntawm tshav kub.

Daim ntawv thov

Tranexamic Acid, tseem hu ua clotting acid, yog ib qho antifibrinolytic amino acid, uas yog ib qho ntawm feem ntau siv tshuaj tua kab mob hauv tsev kho mob.

Cov khoom no tuaj yeem siv rau:

1. Kev raug mob los yog phais los ntshav ntawm prostate, urethra, ntsws, hlwb, tsev menyuam, qog adrenal, thyroid, daim siab thiab lwm yam kabmob uas muaj cov plasminogen activator.

2. Lawv yog siv los ua cov tshuaj thrombolytic, xws li cov ntaub so ntswg plasminogen activator (t-PA), streptokinase thiab urokinase antagonist.

3. Induced rho menyuam, placental exfoliation, stillbirth thiab amniotic kua embolism tshwm sim los ntawm fibrinolytic los ntshav.

4. Menorrhagia, anterior chamber hemorrhage thiab hnyav epistaxis nrog nce hauv zos fibrinolysis.

5. Nws yog siv los tiv thaiv lossis txo los ntshav tom qab kev tshem tawm cov hniav los yog qhov ncauj phais hauv cov neeg mob hemophilic uas muaj qhov muaj VIII lossis yam IX tsis txaus.

6. Cov khoom no yog superior rau lwm cov tshuaj antifibrinolytic nyob rau hauv hemostasis ntawm me me hemorrhage tshwm sim los ntawm rupture ntawm central aneurysm, xws li subarachnoid hemorrhage thiab intracranial aneurysm hemorrhage. Txawm li cas los xij, kev saib xyuas yuav tsum tau them rau qhov kev pheej hmoo ntawm cerebral edema lossis cerebral infarction. Raws li rau cov neeg mob hnyav nrog kev phais mob, cov khoom no tsuas yog siv los ua ib qho tshuaj ntxiv.

7. Rau kev kho mob ntawm hereditary vascular edema, tuaj yeem txo tus naj npawb ntawm kev tawm tsam thiab mob hnyav.

8. Cov neeg mob hemophilia muaj cov ntshav tawm.

9. Nws muaj tseeb curative nyhuv ntawm chloasma.


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